I know this is not in the correct forum section, but I don't think I can post in the more clinical oriented areas of the board.

I had an interesting question on an exam which asked what the rationale of the <2.5 sodium intake rule after an acute MI was.

There were two possibly correct answers: reduction in preload or reduction in afterload. My thinking was that an overall drop in Na+ would decrease both preload and afterload... since a similar effect is seen with loop diuretics as they decrease systemic fluid volume. Between preload and afterload, I'm guessing a reduction in afterload is more important after an acute MI, since it decreases cardiac workload and Mv02.

Haven't seen the answer yet... any thoughts?